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RE: Intercellular Homeostasis

in #intercellular7 hours ago (edited)

Fish Oil
Krill

CH3
Methyl Doner

Phospholipids
Phosphatidylcholine

The body modifies triglycerides (TAG) into phospholipids (specifically phosphatidylcholine, or PC) primarily through a pathway that involves removing a fatty acid, introducing a phosphate-containing group, and utilizing methyl donors (choline/betaine/methionine) to complete the structure.

The body converts triglycerides to phospholipids primarily in the liver and cells through a process called de novo phospholipid synthesis (Kennedy pathway), where a specific lipase enzyme removes one fatty acid from a triglyceride (or diglyceride), and a phosphate group (linked to a nitrogenous base like choline) is added.

Vitamin B12 and Folate support the methionine cycle, which is required to create phosphatidylcholine in the liver.

Methyl Donors (Choline, Betaine, Folate): These nutrients provide the necessary CH3 groups to remethylate homocysteine back into methionine, which then regenerates SAM.

Homocysteine Remethylation: Homocysteine must be remethylated to methionine to prevent toxicity and maintain methylation potential. This occurs via two main pathways:

Betaine Pathway (BHMT): Primarily in the liver, betaine (derived from choline) transfers a methyl group directly to homocysteine.

Folate Pathway (MS): 5-methyltetrahydrofolate (5-MTHF, derived from folate) remethylates homocysteine in a vitamin 𝐵12-dependent reaction.