Reducing the rate at which the infection spreads increases the availability of medical resources to treat those that need it. Flattening the curve reduces the CFR and more people survive.
If 100 people are going to get a disease, and there are only 10 beds available, if all 100 get it at the same time, 90 of them will not get a bed. If 20 of them get it a month for 5 months, half of them will get a bed.
As to the ACE2 issue, the study - the only study I have seen which is supportive of this theory - was not peer reviewed, had exactly 8 subjects to base conclusions on, one of which was an Asian male, and none of which were Caucasian males. What is the basis for concluding from this study that Asians are more susceptible to SARS2 than Caucasians? It's nothing but speculation.
While we can't trust official numbers, since no one but Singapore and South Korea have carefully tracked infections and outcomes, which doesn't affect this issue, today Europe has surpassed all of Asia, including both China and Iran, in officially reported deaths from SARS2. Information from both Iran and Italy indicate they have rates of infection and CFR there, where populations are primarily Caucasian, that are higher than those reported in any ethnically Asian country.
Available evidence, as sketchy as it is, and utterly incapable of being trusted, reveals that the ethnic targeting theory regarding ACE2 expression is backwards. From the data we now see regarding infections and deaths on the ground, Caucasians are more susceptible to infection and fatality from SARS2 than Asians.
In fact, we just have no evidence for any such ethnic targeting or difference for SARS2, because there's no good evidence at all.
A couple weeks ago I read an article on Breitbart that claimed a researcher in Beijing had been convicted and jailed for selling more than $1m worth of used lab animals in a wet market. There was no link to source in the article, and I searched for that source. I found dozens of articles that repeated the claim, simply copy and paste from Breitbart, but no source.
Sometimes people make shit up on the internet for reasons. This ACE2 ethnic targeting theory appears to be exactly that, although the study posted on BiorXiv at least exists, despite not providing evidence supporting the actual ethnic difference in ACE2 expression due to it's lack of participants. You just can't extrapolate from one individual an ethnic difference common to billions of people from an ethnic group that wasn't even represented in the study at all.
I have posted a link to the original study on BiorXiv in the OP, and you can read it for yourself, which I recommend you do if you want to actually understand the basis for the claimed ethnic targeting. There is no nominal evidence whatsoever for that claim, and current infection and fatality rates in predominantly Caucasian nations indicate the opposite targeting has more evidence.
While the absence of evidence is not evidence of absence, it's absence of evidence, and can't support extraordinary conclusions, which require extraordinary evidence.
I wasn't aware the one study with only eight participants was the only ace2 study. For some reason I was under the impression this factor was common knowledge. I think both Boyle and Adams were talking about ace2 matter of factually and if they did so based on a 8 participant study. Ehh, I doubt they both would fall for something like that?
This is why I discussed the Breitbart article. They don't cite sources. Look for sources. If there are no sources other than their unsupported claims, it's not reasonable to believe their unsupported claims IMHO.
I can't find any other sources for this claim, and I've looked. If you can find sources other than the study I linked in the OP, I'd really appreciate a link.
Even outside of the possibility that ACE2 may or may not be race specific. If you catch the HIV flu and you're shedding HIV. Basically, everyone will be shedding HIV. Everyone will get the HIV regardless of how well an individual shelters. One day they'll come into contact with an asymptomatic HIV disease bag who doesn't know they're shedding the virus. The whole thing seems basically impossible to avoid.
This is factually incorrect. It is not unstoppable. There are functional procedures that have been shown to prevent infection, otherwise there wouldn't be a doctor or nurse left in the world that would treat victims.
Treat everyone and yourself as if they were infected, and undertake the necessary procedures to prevent spreading the infection. It's up to us - not our overlords - who survives the pandemic, and the totalitarian aftermath of the oppressive quarantines too.
I guess we shall see what it do, but if a random sneeze or rub of the eyes or touch of the face can give ya HIV. It's just stupid, we'll have to cure HIV.
I think there is a cure: DRACO. It works by finding viral RNA actively being replicated in a cell, and triggering the cell to die. This ends the viral infection. So far it's been tested on more than a dozen RNA viruses and killed them all in vitro. However, a broad spectrum cure for viral diseases is a total destruction of Big Pharma's business model selling vaccines and prescriptions for incurable diseases. They won't fund it, and they also control government funding agencies through graft and corruption, so in vivo testing won't be funded by NIH or CDC either.
Todd Rider has even tried to get crowdfunding to push DRACO further towards approval for public use, but hit the wall. Bill Gates and his buddies don't need funding from other sources to pay for in vivo testing.
I think banksters have the cure. I don't think they're gonna share. I think they want us dead, or sterilized if we live, and better if we have AIDS.
Search DRACO on the duck, or check my backlog for links.
I found some further information outside
of the study with eight participants that
suggest ace-2 is involved.
Scientists figure out how new coronavirus breaks into human cells
https://www.livescience.com/how-coronavirus-infects-cells.html
Researchers at Westlake University Unveiled Structure of the RBD-ACE2-B0AT1 Complex
https://en.westlake.edu.cn/news_events/westlakenews/202002/t20200225_3069.shtml
"Structural basis for the recognition of the SARS-CoV-2 by full-length human ACE2"
https://science.sciencemag.org/content/early/2020/03/03/science.abb2762
Apparently, ibuprofen might increase ACE-2, so there is some chatter about that being a bad idea.
https://www.thelancet.com/action/showPdf?pii=S2213-2600%2820%2930116-8
Yes, the ACE2 receptor is how the virus gets into human cells. But, none of those papers discusses ethnic differences in ACE2 expression, which is what I haven't found any support for, and is what is behind claims it is an ethnically targeted bioweapon. It may be a bioweapon, and I'm more convinced than ever it was at least bioengineered in a lab, but that's not saying it's ethnically targeted.
Dunno if you saw it, but I posted about Shi Zhengli who tinkered with bat SARS-like viruses at the Wuhan Institute of Virology by adding the SARS ability to enter human cells with the ACE2 receptor. She also found adding chunks of HIV to them 'interesting'.
I posted about her and her work at WIV recently.
Good finds regarding ACE2 and susceptibility to SARS2.
Thanks!